Shibasaki H   Imamura M   Arima S   Tanihata J   Kuraoka M   Matsuzaka Y   Uchiumi F   Tanuma S   Takeda S   
Kuraoka M   Nitahara-Kasahara Y   Tachimori H   Kato N   Shibasaki H   Shin A   Aoki Y   Kimura E   Takeda S   
Lim KRQ   Echigoya Y   Nagata T   Kuraoka M   Kobayashi M   Aoki Y   Partridge T   Maruyama R   Takeda S   Yokota T   
Molecular therapy : the journal of the American Society of Gene Therapy 27(1) 76-86 2018年10月 [査読有り]
Janek Hyzewicz   Jun Tanihata   Mutsuki Kuraoka   Yuko Nitahara-Kasahara   Teiva Beylier   Urs T. Ruegg   Axel Vater   Shin'ichi Takeda   
AMERICAN JOURNAL OF PATHOLOGY 187(5) 1147-1161 2017年5月 [査読有り]
Inflammatory events occurring in dystrophic muscles contribute to the progression of Duchenne muscular dystrophy (DMD). Low-intensity training (LIT) attenuates the phenotype of mdx mice, an animal model for DMD. Therefore, we postulated that LIT c...
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA 114(16) 4213-4218 2017年4月 [査読有り]
Duchenne muscular dystrophy (DMD) is a lethal genetic disorder caused by an absence of the dystrophin protein in bodywide muscles, including the heart. Cardiomyopathy is a leading cause of death in DMD. Exon skipping via synthetic phosphorodiamida...